Cold shock represents a fundamental state of cellular imbalance that affects plant physiology at multiple levels. Metabolic enzymatic rates fall asymmetrically, membranes phase-separate, redox reactions stutter, and reactive oxygen species (ROS) accumulation accelerates (Schwarzländer & Finkemeier, 2013). Mitochondria in these conditions are clear targets, as electron transport slows, ROS surge, and ATP homeostasis is destabilized. The speed at which metabolic fluxes are redistributed, vulnerable processes are protected, and energy budgets are recalibrated will determine a cell's fate.

A mitochondrial gatekeeper for cold resilience: deciphering the importance of a structural variant of VDAC1/3a in Solanum habrochaites

Vita F.;
2026-01-01

Abstract

Cold shock represents a fundamental state of cellular imbalance that affects plant physiology at multiple levels. Metabolic enzymatic rates fall asymmetrically, membranes phase-separate, redox reactions stutter, and reactive oxygen species (ROS) accumulation accelerates (Schwarzländer & Finkemeier, 2013). Mitochondria in these conditions are clear targets, as electron transport slows, ROS surge, and ATP homeostasis is destabilized. The speed at which metabolic fluxes are redistributed, vulnerable processes are protected, and energy budgets are recalibrated will determine a cell's fate.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11586/578560
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