ST2L and sST2 inverse relationship supports the pathogenetic hypothesis that ST2L/IL33 axis could drive the mechanism of plaque development and eventually rupture. Correlation with leukocytes provides a further strong evidence confirming inflammatory pathogenesis of atherosclerosis, but most of all leukocytes’ high value could be associated to a greater plaque instability.
ST2 LIGAND AND SOLUBLE ST2 RELATIONSHIP IN THE CAROTID PLAQUE: A STUDY ON 76 CASES UNDERWENT ENDARTERECTOMY
Ciccone, Marco MatteoWriting – Original Draft Preparation
;Scicchitano, PietroWriting – Review & Editing
;Santoro, AnnaritaMembro del Collaboration Group
;Zito, AnnapaolaMembro del Collaboration Group
;Cristina, GaleandroMembro del Collaboration Group
;Angiletta, DomenicoMembro del Collaboration Group
;Pulli, RaffaeleMembro del Collaboration Group
;Marzullo, AndreaWriting – Original Draft Preparation
2019-01-01
Abstract
ST2L and sST2 inverse relationship supports the pathogenetic hypothesis that ST2L/IL33 axis could drive the mechanism of plaque development and eventually rupture. Correlation with leukocytes provides a further strong evidence confirming inflammatory pathogenesis of atherosclerosis, but most of all leukocytes’ high value could be associated to a greater plaque instability.File in questo prodotto:
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