ST2L and sST2 inverse relationship supports the pathogenetic hypothesis that ST2L/IL33 axis could drive the mechanism of plaque development and eventually rupture. Correlation with leukocytes provides a further strong evidence confirming inflammatory pathogenesis of atherosclerosis, but most of all leukocytes’ high value could be associated to a greater plaque instability.

ST2 LIGAND AND SOLUBLE ST2 RELATIONSHIP IN THE CAROTID PLAQUE: A STUDY ON 76 CASES UNDERWENT ENDARTERECTOMY

Ciccone, Marco Matteo
Writing – Original Draft Preparation
;
Scicchitano, Pietro
Writing – Review & Editing
;
Santoro, Annarita
Membro del Collaboration Group
;
Zito, Annapaola
Membro del Collaboration Group
;
Cristina, Galeandro
Membro del Collaboration Group
;
Angiletta, Domenico
Membro del Collaboration Group
;
Pulli, Raffaele
Membro del Collaboration Group
;
Marzullo, Andrea
Writing – Original Draft Preparation
2019-01-01

Abstract

ST2L and sST2 inverse relationship supports the pathogenetic hypothesis that ST2L/IL33 axis could drive the mechanism of plaque development and eventually rupture. Correlation with leukocytes provides a further strong evidence confirming inflammatory pathogenesis of atherosclerosis, but most of all leukocytes’ high value could be associated to a greater plaque instability.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11586/271839
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